Effects of lead exposure on the human body and health implications. OBJECTIVE: To review the literature concerning the risks associated with exposure to lead and lead compounds, especially in children and in populations that are occupationally exposed. In recent years, epidemiologic studies have focused primarily on the neurotoxic effects of lead on children, particularly in terms of impaired intellectual ability and behavioral problems. However, there is still insufficient information on the mechanisms of action that account for such toxicity. More in-depth studies are also needed on the effects of lead exposure on bone, the central nervous system, the cardiovascular system, the kidneys, the liver, the male and female reproductive systems, and the endocrine system.
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Published on Oct 31, SlideShare Explore Search You. Submit Search. Successfully reported this slideshow. We use your LinkedIn profile and activity data to personalize ads and to show you more relevant ads. You can change your ad preferences anytime. Anemia aplasica adquirida.
Upcoming SlideShare. Like this presentation? Why not share! P " Hipol Embed Size px. Start on. Show related SlideShares at end. WordPress Shortcode. Cindy Ledesma , Haematologist Follow. Full Name Comment goes here. Are you sure you want to Yes No. An eBook reader can be a software application for use on a computer such as Microsoft's free Reader application, or a book-sized computer that is used solely as a reading device such as Nuvomedia's Rocket eBook. Users can purchase an eBook on diskette or CD, but the most popular method of getting an eBook is to purchase a downloadable file of the eBook or other reading material from a Web site such as Barnes and Noble to be read from the user's computer or reading device.
Generally, an eBook can be downloaded in five minutes or less Browse by Genre Available eBooks Be the first to like this. No Downloads. Views Total views. Actions Shares. Embeds 0 No embeds. No notes for slide. Anemia aplasica adquirida 1. Fracaso de la hematopoyesis Falla medular 4. Acquired aplastic anemia. Ann Intern Med ; 6. Current concepts in the pathophysiology and treatment of aplastic anemia.
Blood ; Young NS. American Society of Hematology You just clipped your first slide! Clipping is a handy way to collect important slides you want to go back to later. Now customize the name of a clipboard to store your clips. Visibility Others can see my Clipboard. Cancel Save.
Aplasia medular: Actualización
Skip to search form Skip to main content You are currently offline. Some features of the site may not work correctly. La aplasia medular, segun su etiologia puede ser congenita y adquirida; esta ultima es la mas frecuente. La causa del fallo de la hematopoyesis parece ser multifactorial. Se revisan las causas de aplasia medular adquirida, sus mecanismos fisiopatologicos y se hace enfasis en los mecanismos inmunes, que desempenan un papel central en su fisiopatologia. View PDF.
Myelodysplastic syndromes and diseases with myelodysplastic and myeloproliferative features. Myelodysplastic syndromes MDS represent a heterogeneous group of hematologic disorders characterized by ineffective hematopoiesis and an increased risk of developing acute myeloid leukemia. In this educational article the general aspects of the physiopathology, diagnosis, and histopathological features of MDS and their role in differential diagnosis, classification and prognostic categorization are presented. The importance of clinical and laboratory evaluations, including peripheral blood and bone marrow analyses, including morphology - aspirate and core biopsy, cytogenetics, immunophenotype and careful serial follow-up is emphasized. Definite diagnosis of MDS, especially in low-risk subtypes, should consider the exclusion of disorders with reactive bone marrow alterations, such as viral infections for example HIV.
NCBI Bookshelf. Patrick Keefe ; Syed Rizwan A. Bokhari 2. The ability to treat the condition depends on its particular etiology and typically involves addressing the underlying cause, if one exists, and correcting volumetric, nutritional, or electrolytic deficiencies. Whatever solutes the tubule normally reabsorbs do not get reabsorbed adequately in a patient with this syndrome. There are at least 10 inherited causes that include cystinosis, galactosemia, hereditary fructose intolerance, tyrosinemia, Wilson disease, Lowe syndrome, Dent disease, glycogenosis, mitochondrial cytopathies, and idiopathic.